What will happen if hypercholesterolemia appears in patients with renal
disease? Will renal disease cause hypercholesterolemia? What’s the relationship
between hypercholesterolemia and renal disease?
1. How does kidney disease cause hypercholesterolemia?
Influenced by renal fibrosis degeneration, the renal glomerular filtration
barrier is damaged. A large amount of plsma protein is lost in urine, which
leads to the occurrence of hypoproteinemia. Human beings have a whole
self-regulatory mechanism. When the plasma is lost to an abnormal range, human
beings will adjust the compensation of liver, and begin the process and
combination of protein so as to add the lost proteins. In this case, liver
combines excessive lipoprotein, which leads to the fact that lipoprotein is
transfused into blood. In this case, hypercholesterolemia which is caused by the
lost of large amount of protein comes into being.
2. How does hypercholesterolemia influence kidney disease?
As we know, when kidney problem takes place, there must be insufficiency of
blood and oxygen in the capillaries, which will lead to sclerosis of capillaries
for a long time. In this case, if hypercholesterolemia takes place, it will
worsen the kidney capillary sclerosis and accelerate the renal damage.
What shall we do if we both have hypercholesterolemia and kidney damage? We
should firstly decrease the level of hypercholesterolemia. The therapy we can
choose is such as Chinese Medicine. It contains active substances which is
abstracted from natural herbs and can improve the blood circulation, including
micro-blood circulation. Secondly, the effective medicine can help dissolve the
blood stasis and discharge them out, thus decreasing the level of
hypercholesterolemia fundamentally.
After the level of hypercholesterolemia turn normal, there will be a cleaner
inner environment for the repair of the damaged intrinsic cells in the kidney.
In this condition, we can apply Chinese Medicine once more, the effective
medicines of which can repair the damaged intrinsic cells in the kidney, thus
help rebuilding renal function.
Pioneer of combining traditional Chinese and western medicine treatment of kidney disease
Friday, May 3, 2013
Thursday, May 2, 2013
Gouty Nephropathy Cause, Prevention and Treatment
Gouty Nephropathy is commonly seen in western countries. If left untreated,
it will impair kidneys gradually and finally result in Renal Failure.
Cause
Gouty Nephropathy is the complication of gouty which is caused by high uric acid level. Afterwards, the urate will deposit in kidney tissues thus resulting in inflammation in kidneys.
There are mainly three causes of high uric acid. Firstly, purine metabolism disorder can cause more endogenous uric acid to be produced in blood. Secondly, if you have a large amount of foods high in purine, it will also result in increased uric acid level. Thirdly, the uric acid in body can not be filtered out of body by kidneys normally.
Prevention
1. Keep a low-purine diet
You should limit the foods high in purine such as animal organs, sardine, alga, spinach, cauliflower etc.
2. Drink more water
You should drink 2500~3000 ml water per day and eat more washy fruits. The increased urine output can promote the excretion of uric acid thus relieving the burden to kidneys.
3. Eat more alkaline foods
Alkaline foods can promote the dissolving of the urate.
4. Avoid eating hot and spicy foods
You should avoid eating hot and spicy foods such as alcohol and pepper etc.
Treatment
The inflammation in kidneys is caused by the urate. Therefore, to treat Gouty Nephropathy effectively, the therapy should focus on removing the urate and suppressing the inflammation in kidneys as well as restoring the impaired kidneys
In China, a natural remedy –Chinese Medicine has been applied to treat Gouty Nephropathy and has showed enormous effect.
1. Micro-Chinese Medicine Osmotherapy can suppress the inflammation in kidneys thus stopping more damage to kidneys.
2. It can improve the blood circulation and increase blood supply to kidneys. Therefore, it can promote the excretion of urate.
3. The effective integrants can provide some useful substances thus stimulating the self-repairing of the impaired renal tubule.
If you want to know more details about our therapy and the disease, it is free for you to consult us on line.
Gout Nephropathy
In western countries and America, the morbidity rate of Gout Nephropathy is about 0.3%. According to report from European dialysis and transplant association, kidney failure which is caused by gout takes up 0.6 to 1.0% in all kidney failure. In recent years, in China, with people taking in more protein and purine in food, the rate of Gout Nephropathy increases as well. Most people with Gout Nephropathy are those who are fat, like meat and drinking alcohol.
Male patients are more than females. The average age is 45. Generally, after ten years, gout develops into Gout Nephropathy. If it can be diagnosed at an early stage and treated properly, the hyperuricemia can be controlled and renal function can be protected. A lot of urea acid is in the blood, which forms needle-shaped crystal, thus causing excessive immune reaction. In this case, pain takes place in areas such as joints of figures, feet, ankle and thumbs. Long time gout can spread to pinna. Acute gout can lead to pain, swelling, heat at night, which can even waken patients up.
Gout Nephropathy is caused by too much urea acid in the blood or difficulties in discharging of urea acid. The clinical manifestation of urea acid is such as proteinuria, edema or swelling,urinating at night, high blood pressure or hypertension, increased level of urea acid in blood and damage in renal tubules.
Gout Nephropathy is caused by too much urea acid in the blood or difficulties in discharging of urea acid. The clinical manifestation of urea acid is such as proteinuria, edema or swelling,urinating at night, high blood pressure or hypertension, increased level of urea acid in blood and damage in renal tubules.
The causes of Gout Nephropathy:
Gout Nephropathy can be caused by primary one and secondary one. Most primary Gout Nephropathy is genetic, the way of which is not clear. More and more clinical materials indicate that Gout Nephropathy is closely related with obesity, primary high blood pressure, abnormal blood fat, diabetes and resistance to insulin. Secondary Gout Nephropathy can be called by leukemia, myeloma, wound, excessive exericise, etc.
Cause
Gouty Nephropathy is the complication of gouty which is caused by high uric acid level. Afterwards, the urate will deposit in kidney tissues thus resulting in inflammation in kidneys.
There are mainly three causes of high uric acid. Firstly, purine metabolism disorder can cause more endogenous uric acid to be produced in blood. Secondly, if you have a large amount of foods high in purine, it will also result in increased uric acid level. Thirdly, the uric acid in body can not be filtered out of body by kidneys normally.
Prevention
1. Keep a low-purine diet
You should limit the foods high in purine such as animal organs, sardine, alga, spinach, cauliflower etc.
2. Drink more water
You should drink 2500~3000 ml water per day and eat more washy fruits. The increased urine output can promote the excretion of uric acid thus relieving the burden to kidneys.
3. Eat more alkaline foods
Alkaline foods can promote the dissolving of the urate.
4. Avoid eating hot and spicy foods
You should avoid eating hot and spicy foods such as alcohol and pepper etc.
Treatment
The inflammation in kidneys is caused by the urate. Therefore, to treat Gouty Nephropathy effectively, the therapy should focus on removing the urate and suppressing the inflammation in kidneys as well as restoring the impaired kidneys
In China, a natural remedy –Chinese Medicine has been applied to treat Gouty Nephropathy and has showed enormous effect.
1. Micro-Chinese Medicine Osmotherapy can suppress the inflammation in kidneys thus stopping more damage to kidneys.
2. It can improve the blood circulation and increase blood supply to kidneys. Therefore, it can promote the excretion of urate.
3. The effective integrants can provide some useful substances thus stimulating the self-repairing of the impaired renal tubule.
If you want to know more details about our therapy and the disease, it is free for you to consult us on line.
Gout Nephropathy
In western countries and America, the morbidity rate of Gout Nephropathy is about 0.3%. According to report from European dialysis and transplant association, kidney failure which is caused by gout takes up 0.6 to 1.0% in all kidney failure. In recent years, in China, with people taking in more protein and purine in food, the rate of Gout Nephropathy increases as well. Most people with Gout Nephropathy are those who are fat, like meat and drinking alcohol.
Male patients are more than females. The average age is 45. Generally, after ten years, gout develops into Gout Nephropathy. If it can be diagnosed at an early stage and treated properly, the hyperuricemia can be controlled and renal function can be protected. A lot of urea acid is in the blood, which forms needle-shaped crystal, thus causing excessive immune reaction. In this case, pain takes place in areas such as joints of figures, feet, ankle and thumbs. Long time gout can spread to pinna. Acute gout can lead to pain, swelling, heat at night, which can even waken patients up.
Gout Nephropathy is caused by too much urea acid in the blood or difficulties in discharging of urea acid. The clinical manifestation of urea acid is such as proteinuria, edema or swelling,urinating at night, high blood pressure or hypertension, increased level of urea acid in blood and damage in renal tubules.
Gout Nephropathy is caused by too much urea acid in the blood or difficulties in discharging of urea acid. The clinical manifestation of urea acid is such as proteinuria, edema or swelling,urinating at night, high blood pressure or hypertension, increased level of urea acid in blood and damage in renal tubules.
The causes of Gout Nephropathy:
Gout Nephropathy can be caused by primary one and secondary one. Most primary Gout Nephropathy is genetic, the way of which is not clear. More and more clinical materials indicate that Gout Nephropathy is closely related with obesity, primary high blood pressure, abnormal blood fat, diabetes and resistance to insulin. Secondary Gout Nephropathy can be called by leukemia, myeloma, wound, excessive exericise, etc.
Wednesday, May 1, 2013
Children Diabetic Nephropathy and Control Method
Diabetic Nephropathy is a common complication of children Children Diabetic
Nephropathy. It is also a high risk complication, which may lead to the death of
children. Children Diabetic Nephropathy is due to long time increased blood
sugar. Generally, Children Diabetic Nephropathy means tuberous sclerosis due to
damage caused by high blood sugar. It can be divided into five stages as
follows:
In the first and second stage, there is nearly no clinical symptom. Examination in lab also can not show any abnormal signs. There are only some pathological changes actually.
In the third stage, there is also no symptom in clinic. Through a kind of sensitive examination methods, we can find out that the secretion of microalbumin increases. This is reversible. In other means, after treatment, Children Diabetic Nephropathy in this stage can be treated well.
In the fourth stage, Children Diabetic Nephropathy shows some clinical symptoms. Through conventional examination, we can find out that the protein in urine has increased. In this stage, timely and effective is essential and necessary. Or else, it will develop into the fifth stage.
In the fifth stage, Children with Diabetic Nephropathy will suffer from renal failure. They even need to receive dialysis to discharge the toxins which accumulates in the body. So generally, Children with Diabetes should have an examination about renal function five years after they are diagnosed with Diabetes.
So to control Diabetes and prevent it from developing into Diabetic Nephropathy, children should pay attention to the following aspects.
Firstly, control the blood pressure. As for children with type 1 Diabetes, high blood pressure can easily lead to Diabetic Nephropathy. In turn, high blood pressure can worsen type 1 Diabetes. So before it develops into Diabetic Nephropathy, controlling the blood pressure is very important, especially for those with type 1 Diabetes. If controlled well, the time when Diabetes develops into Diabetic Nephropathy will be delayed 10 to 20 years.
Secondly, children with type 1 Diabetes should control the intake of protein so as to reduce the load of the kidney. When the intake amount of protein is reduced, children can take in more carbohydrate to supply what the body needs.
Last but not least, children with Diabetes should control the blood sugar, which is an essential factor to prevent Diabetes developing into Diabetic Nephropathy.
In the first and second stage, there is nearly no clinical symptom. Examination in lab also can not show any abnormal signs. There are only some pathological changes actually.
In the third stage, there is also no symptom in clinic. Through a kind of sensitive examination methods, we can find out that the secretion of microalbumin increases. This is reversible. In other means, after treatment, Children Diabetic Nephropathy in this stage can be treated well.
In the fourth stage, Children Diabetic Nephropathy shows some clinical symptoms. Through conventional examination, we can find out that the protein in urine has increased. In this stage, timely and effective is essential and necessary. Or else, it will develop into the fifth stage.
In the fifth stage, Children with Diabetic Nephropathy will suffer from renal failure. They even need to receive dialysis to discharge the toxins which accumulates in the body. So generally, Children with Diabetes should have an examination about renal function five years after they are diagnosed with Diabetes.
So to control Diabetes and prevent it from developing into Diabetic Nephropathy, children should pay attention to the following aspects.
Firstly, control the blood pressure. As for children with type 1 Diabetes, high blood pressure can easily lead to Diabetic Nephropathy. In turn, high blood pressure can worsen type 1 Diabetes. So before it develops into Diabetic Nephropathy, controlling the blood pressure is very important, especially for those with type 1 Diabetes. If controlled well, the time when Diabetes develops into Diabetic Nephropathy will be delayed 10 to 20 years.
Secondly, children with type 1 Diabetes should control the intake of protein so as to reduce the load of the kidney. When the intake amount of protein is reduced, children can take in more carbohydrate to supply what the body needs.
Last but not least, children with Diabetes should control the blood sugar, which is an essential factor to prevent Diabetes developing into Diabetic Nephropathy.
Diet after Diabetes Develops into Diabetic Nephropathy
What is the diet after diabetes develops into diabetic nephropathy?
1. Restrict the intake of Protein: long term of high-protein diet can aggravate the condition of high filtration state, lead to increased metabolin of poisonous nitrogen products and cause the retention of those poisonous products, which further damages the injured kidney. So Diabetic Nephropathy patients are suggested to properly restrict the intake of protein, avoiding the condition mentioned above. In order to relieve the burden of kidney, patients should take high quality protein and have a low protein diet, such as lean meat, egg, milk, fish, etc.
2.Energy: when patients take a low protein diet, they should insure the energy supplement of the body to keep the normal physiological requirements.
3. Low-fat diet: patients at end-stage of renal disease (ESRD) can suffer lipoidosis, so they should take low-fat diet such as olive oil or arachis oil, which contains much unsaturated fatty acid.
4. Restrict the intake of salt: patients when they suffer Hypertension or other obvious symptoms such as edema or decreased urine volume, they should restrict the salt intake, which can relieve the symptom. However, when patients have those symptoms such as vomiting, diarrhea, they should not strictly restrict salt or even supplement salt.
5. Water intake: when patients don not have obvious symptom of edema, they should not restrict water and they need some more water to discharge the waste products in body. However, when there are symptoms of obvious edema, oliguria, anuria, patients should restrict water intake. Otherwise, masses of water intake will aggravate burden to kidney. As a result, ESRD patients should restrict water intake according to urine volume. In addition, when patients suffer fever, diarrhea, vomiting, they should supplement more water.
6. Potassium intake: patients should not restrict the intake of potassium when it is in normal range. However, potassium should be restricted when patients suffer Hyperkalemia and when they suffer Hypokalemia patients should supplement potassium. high-potassimum food such as spinach, bananas, fungus, bean products.
Other substances such as calcium, phosphorus: when kidney is damaged, the discharge of phosphorus will be reduced, leading high level phosphorus. In addition, the injured kidney can also lead to the declined generation of vitamin D3, which affects the absorb of calcium. When the calcium in blood is in a low level, it can easily lead to osteoporosis. So the ideal diet should contain more calcium but less phosphorus. A low protein diet can reduce the intake of phosphorus.
More information about diet after diabetes develops into diabetic nephropathy, please communicate with us online or email us.
1. Restrict the intake of Protein: long term of high-protein diet can aggravate the condition of high filtration state, lead to increased metabolin of poisonous nitrogen products and cause the retention of those poisonous products, which further damages the injured kidney. So Diabetic Nephropathy patients are suggested to properly restrict the intake of protein, avoiding the condition mentioned above. In order to relieve the burden of kidney, patients should take high quality protein and have a low protein diet, such as lean meat, egg, milk, fish, etc.
2.Energy: when patients take a low protein diet, they should insure the energy supplement of the body to keep the normal physiological requirements.
3. Low-fat diet: patients at end-stage of renal disease (ESRD) can suffer lipoidosis, so they should take low-fat diet such as olive oil or arachis oil, which contains much unsaturated fatty acid.
4. Restrict the intake of salt: patients when they suffer Hypertension or other obvious symptoms such as edema or decreased urine volume, they should restrict the salt intake, which can relieve the symptom. However, when patients have those symptoms such as vomiting, diarrhea, they should not strictly restrict salt or even supplement salt.
5. Water intake: when patients don not have obvious symptom of edema, they should not restrict water and they need some more water to discharge the waste products in body. However, when there are symptoms of obvious edema, oliguria, anuria, patients should restrict water intake. Otherwise, masses of water intake will aggravate burden to kidney. As a result, ESRD patients should restrict water intake according to urine volume. In addition, when patients suffer fever, diarrhea, vomiting, they should supplement more water.
6. Potassium intake: patients should not restrict the intake of potassium when it is in normal range. However, potassium should be restricted when patients suffer Hyperkalemia and when they suffer Hypokalemia patients should supplement potassium. high-potassimum food such as spinach, bananas, fungus, bean products.
Other substances such as calcium, phosphorus: when kidney is damaged, the discharge of phosphorus will be reduced, leading high level phosphorus. In addition, the injured kidney can also lead to the declined generation of vitamin D3, which affects the absorb of calcium. When the calcium in blood is in a low level, it can easily lead to osteoporosis. So the ideal diet should contain more calcium but less phosphorus. A low protein diet can reduce the intake of phosphorus.
More information about diet after diabetes develops into diabetic nephropathy, please communicate with us online or email us.
Diet for Diabetes 7 Years, Creatinine 1.6
If creatinine is 1.6, it indicates that diabetes has damaged kidney for in
normal condition, the creatinine level is 0.6 to 1.2. Here are some suggestions
on daily diet.
1.Low potassium diet, do not eat food containing high-level potassium like edible fungus, potato, sweet potato, Chinese cabbage, dates, banana, orange, yiner (white fungus), peach, apricot,spinach.
2.low salt diet ,do not eat things like soda biscuit , soy sauce, steamed oily sticks, especially for patients who has swelling symptoms and unstable hypertension .
3.Low-phosphate diet ,do not eat seafood and plant in the sea, kelp, dried purple seaweed ,animal abdomen organs, sesame, tea, honey, egg yolk.
4.Low fat diet. Do not eat fat, greasy food, fast food, use vegetable oil instead of animal oil when cooking.
5.High-quality low-protein food. Vegetable oil is not advisable, do not eat bean products like tofu, soy bean, milk powder, sunflower seeds, walnut, peanut and dried fruit. You can have a proper amount of egg white, and milk.
6.High vitamin diet. You can eat food rich in fiber and fruits like pear, apple, cucumber,tomato, etc.
7.No spicy food (ginger, onion, garlic). No smoking.
Last but not least, patients should keep low sugar diet or else, it will worsen blood sugar. What’s more, patients will diabetes should also monitor blood sugar. Low blood sugar is more serious, which may even threaten people’s life.
1.Low potassium diet, do not eat food containing high-level potassium like edible fungus, potato, sweet potato, Chinese cabbage, dates, banana, orange, yiner (white fungus), peach, apricot,spinach.
2.low salt diet ,do not eat things like soda biscuit , soy sauce, steamed oily sticks, especially for patients who has swelling symptoms and unstable hypertension .
3.Low-phosphate diet ,do not eat seafood and plant in the sea, kelp, dried purple seaweed ,animal abdomen organs, sesame, tea, honey, egg yolk.
4.Low fat diet. Do not eat fat, greasy food, fast food, use vegetable oil instead of animal oil when cooking.
5.High-quality low-protein food. Vegetable oil is not advisable, do not eat bean products like tofu, soy bean, milk powder, sunflower seeds, walnut, peanut and dried fruit. You can have a proper amount of egg white, and milk.
6.High vitamin diet. You can eat food rich in fiber and fruits like pear, apple, cucumber,tomato, etc.
7.No spicy food (ginger, onion, garlic). No smoking.
Last but not least, patients should keep low sugar diet or else, it will worsen blood sugar. What’s more, patients will diabetes should also monitor blood sugar. Low blood sugar is more serious, which may even threaten people’s life.
Monday, April 22, 2013
Why Do I Have Nausea and Tiredness after Dialysis
Many patients may have nausea and tiredness after dialysis treatment. These
terrible feelings may make the patients not want to have another dialysis
treatment. Why the patients experience those horrible feelings?
Usually, the bad feelings will disappear in a few hours and by the next day. If not, the patients should make out if it is related to another problem.
It can help the patients on dialysis stop this from happening to know the causes of tiredness and nausea after dialysis. The detailed discussion of each reason are as follows:
Low blood pressure
Low blood pressure is the most common cause of tiredness and nausea. During dialysis treatment, fluid is removed to eliminate the building up of extra fluid in body. As healthy kidneys work in 24 hours persistently to remove the fluid out of body, the fluid is required to be excreted out of body in a short time. It is very likely to cause low blood pressure.
Severe aneamia
Severe aneamia is commonly seen in Renal Failure and can cause low blood pressure and weakness. This can be magnified by dialysis treatment.
Infection
Infection can lead to low blood pressure, nausea and tiredness. This can happen if there is a new infection that happens during dialysis or if there is a chronic infection that affects the way the body can tolerate the dialysis session.
Blood pressure medications
Many patients with Renal Failure have high blood pressure. Dialysis can lower blood pressure by removing extra fluid so most patients are told to stop medications for blood pressure during dialysis. As the medications start working and lowering blood pressure and the machine is removing fluid and lowering blood pressure, the blood pressure may go too low. Therefore, some patients with Renal Failure are recommended to stop taking medications for blood pressure, but some should not. It is necessary for you to discuss it with your doctors before starting dialysis.
The above are the causes of tiredness and nausea after dialysis. There are also some other possible reasons for it such as problem with dry weight, allergic reaction, etc. To learn more detailed information, please consult our online experts.
Usually, the bad feelings will disappear in a few hours and by the next day. If not, the patients should make out if it is related to another problem.
It can help the patients on dialysis stop this from happening to know the causes of tiredness and nausea after dialysis. The detailed discussion of each reason are as follows:
Low blood pressure
Low blood pressure is the most common cause of tiredness and nausea. During dialysis treatment, fluid is removed to eliminate the building up of extra fluid in body. As healthy kidneys work in 24 hours persistently to remove the fluid out of body, the fluid is required to be excreted out of body in a short time. It is very likely to cause low blood pressure.
Severe aneamia
Severe aneamia is commonly seen in Renal Failure and can cause low blood pressure and weakness. This can be magnified by dialysis treatment.
Infection
Infection can lead to low blood pressure, nausea and tiredness. This can happen if there is a new infection that happens during dialysis or if there is a chronic infection that affects the way the body can tolerate the dialysis session.
Blood pressure medications
Many patients with Renal Failure have high blood pressure. Dialysis can lower blood pressure by removing extra fluid so most patients are told to stop medications for blood pressure during dialysis. As the medications start working and lowering blood pressure and the machine is removing fluid and lowering blood pressure, the blood pressure may go too low. Therefore, some patients with Renal Failure are recommended to stop taking medications for blood pressure, but some should not. It is necessary for you to discuss it with your doctors before starting dialysis.
The above are the causes of tiredness and nausea after dialysis. There are also some other possible reasons for it such as problem with dry weight, allergic reaction, etc. To learn more detailed information, please consult our online experts.
How to treat Focal Segmental Glomerulosclerosis (FSGS)
What’s is Focal Segmental Glomerulosclerosis (FSGS)? How to treat Focal
Segmental Glomerulosclerosis (FSGS)? Is it difficult to treat Focal Segmental
Glomerulosclerosis (FSGS)? These questions are always confusing patients with
FSGS. In western countries, patients often adopt hormone, which has not
satisfactory effect for it is not allergic to hormone, and difficult to treat.
In this passage, doctors in our Hospital will introduce for you what is Focal
Segmental Glomerulosclerosis (FSGS) and the characteristic treatment in our
hospital.
Why do people get Focal Segmental Glomerulosclerosis (FSGS)? Actually, the reason can be divided into two kinds: renal reasons such as low immunity and other reasons such as infections and cold, which leads inflammatory cells to invade and to combine with mesangial cell. As a result, a large amount of immune complex such as C3、lgG、lgM come into being and deposite in renal glomerular capillary loops, and mesangial area, which leads to the damage of mediated immune in mesangial cells of blood capillary, which damages the normal function of renal glomerular mesangial cells. In addition, the premier function which contains combined discharge barrier is also damaged.
After this function is damaged, protein in blood which shows negative charge will also be damaged due to the damage of barrier in mesangial cell. The protein will also discharge together with urine. In clinic, the urinary examination will show three pluses or two pluses.
From the above, we can make a conclusion that Focal Segmental Glomerulosclerosis (FSGS) is a kind of immune disease, which manifest as protein in urine and a series of discomfort, including edema, etc.
How to treat Focal Segmental Glomerulosclerosis (FSGS) then? Is there a much better therapy instead of hormone? Traditional Chinese herbal medicine and stem cell transplant can make it.
Firstly, Traditional Chinese herbal medicine contains many nutritious substances, which can play roles after entering patients’ body. Then, the insufficiency of blood and oxygen in mesangial cell and other damaged intrinsic cells can be improved, which means that damaged mesangial cell and other damaged intrinsic cells can be repaired. In addition, traditional Chinese medicine has a micro-process compared with traditional Chinese medicine. So in contrast to hormone and other western medicine, traditional Chinese herbal medicine has no side effect, which makes it more popular among more and more patients in China and abroad.
How about stem cell transplant? As for those damaged cell, traditional Chinese herbal medicine can repair it and as for those necrotic ones, stem cell can generate into intrinsic necrotic cells. In addition, stem cell transplant can adjust the immunity.
In a word, the combination of traditional Chinese herbal medicine and stem cell transplant can achieve a perfect effect in treating Focal Segmental Glomerulosclerosis (FSGS).
Why do people get Focal Segmental Glomerulosclerosis (FSGS)? Actually, the reason can be divided into two kinds: renal reasons such as low immunity and other reasons such as infections and cold, which leads inflammatory cells to invade and to combine with mesangial cell. As a result, a large amount of immune complex such as C3、lgG、lgM come into being and deposite in renal glomerular capillary loops, and mesangial area, which leads to the damage of mediated immune in mesangial cells of blood capillary, which damages the normal function of renal glomerular mesangial cells. In addition, the premier function which contains combined discharge barrier is also damaged.
After this function is damaged, protein in blood which shows negative charge will also be damaged due to the damage of barrier in mesangial cell. The protein will also discharge together with urine. In clinic, the urinary examination will show three pluses or two pluses.
From the above, we can make a conclusion that Focal Segmental Glomerulosclerosis (FSGS) is a kind of immune disease, which manifest as protein in urine and a series of discomfort, including edema, etc.
How to treat Focal Segmental Glomerulosclerosis (FSGS) then? Is there a much better therapy instead of hormone? Traditional Chinese herbal medicine and stem cell transplant can make it.
Firstly, Traditional Chinese herbal medicine contains many nutritious substances, which can play roles after entering patients’ body. Then, the insufficiency of blood and oxygen in mesangial cell and other damaged intrinsic cells can be improved, which means that damaged mesangial cell and other damaged intrinsic cells can be repaired. In addition, traditional Chinese medicine has a micro-process compared with traditional Chinese medicine. So in contrast to hormone and other western medicine, traditional Chinese herbal medicine has no side effect, which makes it more popular among more and more patients in China and abroad.
How about stem cell transplant? As for those damaged cell, traditional Chinese herbal medicine can repair it and as for those necrotic ones, stem cell can generate into intrinsic necrotic cells. In addition, stem cell transplant can adjust the immunity.
In a word, the combination of traditional Chinese herbal medicine and stem cell transplant can achieve a perfect effect in treating Focal Segmental Glomerulosclerosis (FSGS).
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